Atrial flutter and atrial fibrillation are two closely related heart rhythm disorders. They can coexist in the same individual, with one or the other appearing at different times over the course of life.

Although it would be possible to group them into a single text because of their similarities, they are presented separately to allow a clearer understanding of their specific mechanisms, clinical features, and treatment options.

Often rapid heartbeats

Atrial flutter is an arrhythmia characterized by a generally rapid and regular heart rhythm.

The heart rate may exceed 150 beats per minute, which explains the frequent perception of palpitations.

An arrhythmia originating in the atria

Like atrial fibrillation, atrial flutter originates in the atria, the upper chambers of the heart.

An overexcited new conductor

In atrial flutter, the heart’s usual electrical conductor is replaced by an abnormal electrical circuit, responsible for this excessive atrial activity.

Fortunately, the electrical conduction system between the atria and the ventricles acts as a filter: only one impulse out of two is transmitted to the ventricles. This results in a pulse often close to 150 beats per minute.

Thus, as in atrial fibrillation, the heart rate is rapid. However, unlike atrial fibrillation, the pulse most often remains regular.

The heart rate may be slower when the conduction system is already impaired or under the effect of medications intended to slow the heart.

Risk of blood clot formation in the heart

The main risk associated with atrial flutter is the formation of blood clots that can enter the circulation.

When the atria beat at a very high rate, around 300 beats per minute, their contraction becomes ineffective. Blood then tends to pool within the atria, which promotes clot formation, a phenomenon also seen in atrial fibrillation.

The major danger is that a clot can break loose and be carried into the systemic circulation, causing a systemic embolism. If a fragment travels to the brain, it can lead to a stroke, the consequences of which may be severe and sometimes permanent.

A generally benign arrhythmia

Despite the possible risk of blood clot formation, atrial flutter is generally considered a benign arrhythmia, especially when it is well managed and appropriately monitored.

IT IS POSSIBLE TO LIVE VERY WELL WITH ATRIAL FLUTTER

Absence of symptoms or variable palpitations

In some individuals, atrial flutter may be completely asymptomatic, with no perception of palpitations.
Others experience rapid and uncomfortable heartbeats, sometimes causing anxiety.

Some people instead report:

  • unusual fatigue,
  • a reduced ability to perform physical activity,
  • shortness of breath, particularly during exertion.

 

The intensity of symptoms therefore varies greatly from one person to another.

Possible causes of atrial flutter

The prevalence of atrial flutter increases with age, which explains why it is more commonly observed in older adults. Depending on the populations studied, it may affect approximately 5 to 10% of individuals.

Certain medical conditions are more frequently associated with its occurrence, including:

  • hypertension,
  • diabetes,
  • heart failure.

As with atrial fibrillation, other diseases or situations may also promote this rhythm disorder, such as:

  • vascular conditions,
  • diseases of the heart muscle or the coronary arteries,
  • inflammation of the lining of the heart (pericarditis), particularly after cardiac or thoracic surgery,
  • certain lung diseases, such as chronic obstructive pulmonary disease (COPD) or pulmonary embolism,
  • thyroid disorders, especially hyperthyroidism.

 

In some cases, however, no specific cause can be identified. This is referred to as idiopathic atrial flutter.

Two main treatment approaches

Treatment of atrial flutter is based on two primary strategies:

  • Rhythm control, which aims to stop the arrhythmia and restore a normal heart rhythm, known as sinus rhythm.
  • Rate control, which focuses on slowing the heart rate without necessarily eliminating the arrhythmia.

 

-An important evolution in management

For many years, catheter ablation was mainly proposed when medications were no longer sufficient to control atrial flutter or caused significant side effects. As a result, this procedure was often considered late in the course of the condition.

Over time, clinical experience and improvements in technique have changed this approach. It is now recognized that, in certain individuals—particularly younger patients who are otherwise healthy—earlier intervention may offer long-term benefits. The goal is no longer limited to symptom relief, but also to reduce the long-term impact of atrial flutter on the heart.

In this context, catheter ablation may be considered earlier, sometimes even before the onset of significant symptoms. This strategy aims to reduce the persistence of the arrhythmia, lower the risk of future complications, and improve the overall course of the condition.

It is important to emphasize, however, that this decision is never automatic. The choice of treatment is always based on an individualized assessment, taking into account age, the duration of atrial flutter, overall heart health, associated medical conditions, and personal preferences.

The decision is made in collaboration with the medical team in order to offer the most appropriate option for each situation.

1- Ablation: treating the arrhythmia at its source

When catheter ablation is chosen, the goal is to directly treat the mechanism responsible for atrial flutter.

This procedure may be proposed at the initial evaluation or after failure or intolerance of previous treatments, particularly when symptoms are significant or persistent.

During the procedure, the areas within the atria responsible for the arrhythmia are precisely identified. They are then neutralized using:

  • radiofrequency energy (heat),
  • cryotherapy (cold),
  • or pulsed field energy.

 

By targeting the source of the rhythm disorder itself, this approach often results in a durable elimination of the arrhythmia.

→ To read: Ablation of an arrhythmia

2- When ablation is not chosen initially

Although catheter ablation may be considered earlier in some individuals, it is not always the first option.

Other ways of treating atrial flutter may be entirely appropriate, depending on the clinical situation and how the arrhythmia manifests.

A) Controlling the heart rate

In people with few or no symptoms, atrial flutter can often be managed by slowing the heart rate with medication. This approach is still frequently considered as a first-line option, particularly in older patients.

In some cases, however, these medications may slow the heart excessively. When this occurs, the discussion may include the possibility of implanting a permanent pacemaker. This device acts as an electrical safety net, preventing the heart rate from dropping below an inadequate level.

B) Controlling the heart rhythm with medication

In some individuals, even when ablation is not selected as a first option, persistent palpitations may significantly affect quality of life. In this context, another strategy is to attempt to restore and maintain a more regular heart rhythm using medication.

Two medication-based approaches may be considered.

The first involves taking a medication daily to prevent episodes of atrial flutter. This option is generally proposed when episodes are frequent or prolonged.

The second approach, sometimes referred to as “pill-in-the-pocket,” consists of taking medication only at the onset of symptoms.

This strategy is mainly used in people whose episodes are infrequent, well tolerated, and occur sporadically. It helps limit daily medication use while still providing a way to stop the arrhythmia when it occurs.

In all cases, treatment effectiveness and tolerance vary from one person to another. The choice of strategy is based on an individualized evaluation and regular medical follow-up.

Electrical cardioversion

In some situations, a normal heart rhythm is not restored despite the initiation of antiarrhythmic medication. When this occurs, another strategy may be considered. It involves delivering a controlled electrical shock to the chest in order to “reset” the heart’s electrical system. This brief electrical pause then allows the heart to resume a normal rhythm under the guidance of its natural “conductor.”

This procedure is known as electrical cardioversion.

Contrary to the image often portrayed on television, cardioversion is neither dramatic nor violent. The body does not jerk as shown on screen, and the procedure takes place in a controlled medical environment.

Electrical cardioversion requires a short period of anesthesia. Once the person is fully asleep, the shock is delivered. The sedation wears off quickly, and awakening is usually smooth.

Like any medical procedure, cardioversion carries certain risks. These are carefully evaluated and explained beforehand, and this option is chosen when the expected benefits outweigh the potential risks.

Reducing the risk of blood clot formation

The role of an anticoagulant is not to “thin the blood,” but rather to reduce the risk of blood clots forming in the heart and to prevent them from traveling through the bloodstream.

The decision to prescribe an anticoagulant is based on an individualized assessment. The physician considers several factors, including:

  • age,
  • the presence of diabetes or high blood pressure,
  • a history of stroke,
  • the presence of heart failure.

 

Once this type of treatment is started, it is often required long term, sometimes even indefinitely.
At each step, the physician carefully balances the risk of embolization (formation and migration of a clot) against the risk of bleeding, in order to choose the safest strategy.

Never stop anticoagulant medication without medical advice

Anticoagulant therapy should never be stopped on one’s own initiative.

Any change or interruption must be discussed with the physician, as inappropriate discontinuation may expose the individual to a significantly increased risk of serious complications, including stroke.

ANTICOAGULANTS ARE NOT INTENDED TO THIN THE BLOOD, BUT TO PREVENT THE FORMATION OF BLOOD CLOTS.

Conclusion

Atrial flutter is a common heart rhythm disorder that is generally benign, but it requires appropriate management because of the potential risk of blood clot formation and stroke.
Its clinical presentation varies widely: some individuals experience no symptoms, while others may be troubled by palpitations, fatigue, or shortness of breath.

Treatment is based on two complementary approaches: controlling the heart rhythm and preventing blood clots, using strategies tailored to each situation. With medication, cardioversion, and, in some cases, catheter ablation, it is now possible to effectively control this arrhythmia and maintain a good quality of life.

Regular medical follow-up remains essential to adjust treatment, prevent complications, and ensure safe long-term management.